Ice cream headache | |
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The trigeminal nerve, shown in yellow, conducts signals from dilating blood vessels in the palate to the brain, which interprets the pain as coming from the forehead. | |
Classification and external resources | |
Specialty | neurology |
ICD-10 | G44.8021 (ICD-10NA) |
An ice-cream headache, also known as brain freeze, cold-stimulus headache, trigeminal headache or its given scientific name sphenopalatine ganglioneuralgia (meaning "nerve pain of the sphenopalatine ganglion"), is a form of brief pain or headache commonly associated with consumption (particularly quick consumption) of cold beverages or foods such as ice cream and ice pops. It is caused by having something cold touch the roof of the mouth (palate), and is believed to result from a nerve response causing rapid constriction and swelling of blood vessels or a "referring" of pain from the roof of the mouth to the head. The rate of intake for cold foods has been studied as a contributing factor. Ice-cream headache is distinct from dentin hypersensitivity, a type of pain that can occur under similar circumstances.
Cats and other animals have been observed experiencing a similar reaction when presented with a similar stimulus.
The term ice-cream headache has been in use since at least January 31, 1937, contained in a journal entry by Rebecca Timbres published in the 1939 book We Didn't Ask Utopia: A Quaker Family in Soviet Russia.
An ice cream headache is the direct result of the rapid cooling and rewarming of the capillaries in the sinuses leading to periods of vasoconstriction and vasodilation. A similar but painless blood vessel response causes the face to appear "flushed" after being outside on a cold day. In both instances, the cold temperature causes the capillaries in the sinuses to constrict and then experience extreme rebound dilation as they warm up again.
In the palate, this dilation is sensed by nearby pain receptors, which then send signals back to the brain via the trigeminal nerve, one of the major nerves of the facial area. This nerve also senses facial pain, so as the neural signals are conducted the brain interprets the pain as coming from the forehead—the same "referred pain" phenomenon seen in heart attacks. Brain-freeze pain may last from a few seconds to a few minutes. Research suggests that the same vascular mechanism and nerve implicated in "brain freeze" cause the aura (sensory disturbance) and pulsatile (throbbing pain) phases of migraines.